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J Am Dent Assoc, Vol 131, No 5, 623-632.
© 2000 American Dental Association

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CLINICAL PRACTICE

TREATMENT OPTIONS FOR IMPACTED TEETH



CHARLES A. FRANK, D.M.D., M.S.


   ABSTRACT
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
Background. A compilation of treatment options for impacted teeth is presented to assist dentists in discussing the sequelae of impacted teeth as well as the complications of treatment with their patients.

Overview. A differential diagnosis for an impacted tooth is not possible without clinical assessment. However, an ankylosed tooth or a tooth with failure of its eruption mechanism may be mistaken for an impacted tooth. This review of national and international dental texts, journals and publications concerning impacted teeth spans more than 50 years.

Practice Implications. Treatment options for the management of impacted teeth are separated into four categories: observation, intervention, relocation and extraction. The indications, contraindications and complications of each option are presented. This information will help the clinician identify developmental conditions associated with an increased probability of tooth impaction and assess available treatment options.

The intent of this article is to consolidate and organize available information regarding treatment for impacted teeth and the associated complications.

Studies have reported that the incidence of tooth impaction varies from 5.6 to 18.8 percent of the population.15 Any permanent tooth may become impacted; however, the maxillary canine, with the exception of the third molars, has been the most frequently impacted tooth.15 Although impacted teeth (other than third molars) affect a small percentage of the population, they accounted for 22.4 percent of patients referred for orthodontic evaluation in a Swedish study.4


   TREATMENT OPTIONS
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
There are four treatment options for impacted teeth: observation, intervention, relocation and extraction. Some interaction among treatment options is likely. Observation implies no treatment for a specific period, which is subdivided into preimpaction and postimpaction periods. Intervention consists of a brief period of orthodontic therapy or the removal of teeth (deciduous and/or permanent), with the attempt to eliminate tooth impaction. Relocation refers to the repositioning of an impacted tooth surgically or orthodontically. Extraction in this report refers only to the removal of an impacted permanent tooth.


   OBSERVATION
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
Observation of the dentition normally begins with the completion of the deciduous dentition eruption and ends with the removal or eruption of an impacted tooth. To be a viable treatment option, a dental professional using clinical and radiographic evaluation can, in most cases, predict tooth impaction (that is, the preimpaction observation period) or anticipate pathological sequelae associated with impacted teeth (that is, the postimpaction observation period). If the deciduous dentition has erupted without incident and findings on the oral examination are unremarkable, the preimpaction observation period begins with the eruption of first permanent molars at approximately 6 years of age.

Preimpaction observation. I present this period chronologically to allow the dentist to assess the development of the dentition at significant times.

Ectopic eruption of molars. The most common ectopically erupting permanent teeth are the maxillary first molars, the maxillary canines and the mandibular lateral incisors.6 Before patients reach age 7 years, researchers and clinicians recommend that ectopically erupting maxillary first molars be observed, because their eruption is still deemed possible.610 After age 7 years, molar eruption is considered impossible. Figure 1Go shows maxillary and mandibular arch-length discrepancies in an 8-year-old boy, as evidenced by the premature loss of the deciduous mandibular right canine and atypical resorption of the deciduous maxillary left canine root by the erupting maxillary left lateral incisor.



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Figure 1. Panoramic radiograph of an 8-year-old boy with maxillary and mandibular arch-length discrepancies. Note the mesial angulation of the ectopically erupted maxillary right first molar.

 
My literature search revealed that maxillary first-molar ectopic eruption is highly correlated with large permanent maxillary teeth, abnormal permanent maxillary first molar angle of eruption, short maxillae, large deciduous maxillary teeth, posterior positioning of the maxilla and delayed calcification of affected maxillary first molars.1012 The mesial inclination of the developing maxillary first molar and its location (apical to the distobuccal root of the deciduous second molar) are radiographic prognosticators for ectopic eruption.8 Untreated irreversible ectopic eruption of the maxillary first molar results in loss of arch length.

Ectopic eruption of incisors. Ectopic eruption of the mandibular lateral incisor may delay its emergence into the mouth or result in the premature loss of the adjacent deciduous canine. Pronounced mandibular incisor crowding or premature loss of one or both deciduous canines is commonly associated with mandibular arch-length discrepancy. Therefore, analysis of arch length should be done.

Supernumerary teeth. About 1 to 3 percent of the population have supernumerary teeth.13,14 More than 90 percent of supernumerary teeth are found in the maxilla, with the vast majority located in the premaxilla.1113,15 Primosch13 recommended routine radiographic examination of the maxillary occlusal surface in children aged 6 through 9 years. Because supernumerary teeth frequently appear in the premaxilla, their presence must be ruled out when maxillary incisor eruption is delayed or the eruption sequence is atypical. Complications associated with the presence of supernumerary teeth are cystic degeneration, nasal eruption, interference with the eruption of adjacent permanent teeth (delayed or displaced), resorption of adjacent permanent teeth, permanent tooth rotation, diastema, local infection and periodontitis.11,13

In an attempt to establish treatment options, authors11,12,14 have categorized supernumerary teeth by their shape: conical (peglike) and tuberculate (tooth width and length are similar).11,12,14 The conical form is the most common, is usually located between the maxillary incisors and frequently erupts. The conical form can displace or rotate adjacent maxillary incisors; however, incisor eruption is rarely delayed.14 Therefore, conical supernumerary teeth are permitted to erupt, which simplifies their removal. The inverted conical form is frequently associated with cystic lesions and can erupt into the nasal floor, becoming more difficult to remove with time.11 The removal of these supernumerary teeth is recommended.13,14 The tuberculate form rarely erupts, is frequently bilateral and interferes with permanent tooth eruption.11,12 Immediate removal of the tuberculate supernumerary tooth is also recommended.13,14

Tissue obstructions. If permanent tooth eruption is delayed and if findings of clinical and radiographic examinations are unremarkable, then the dental professional should consider soft-tissue or osseous interferences to eruption. Non-pathological soft-tissue interferences arise from thickened gingival tissue or thickened follicular tissue.9,16 If the stage of root development is not consistent with the extent of eruption, I recommend that the clinician compare clinical and radiographic findings with those obtained more than six months earlier to assess tooth eruption.

Ectopic eruption of canines. Researchers recommend early detection of impacted maxillary canines because of their unpredictable and rapid resorption of the maxillary incisor roots.1719 Ericson and Kurol17 reported that buccally and palatally impacted canines were proportionally associated with incisor resorption. Resorption has not been associated with canine impaction before age 10 years.18 Ericson and Kurol18,19 found that incisor root resorption was three times more prevalent in girls than in boys. This may be because of more advanced stages of dental development in girls.2023

The eruption path of the maxillary canine is not radiographically predictable before 10 years of age.19,21,22 However, after 10 years of age, alveolar contour was a good predictor of unerupted canine position (especially when compared with the contralateral canine).21,22,24 According to Ericson and Kurol,21 5 percent of 10- to 11-year-old children have unerupted nonpalpable canines. These authors21 recommended radiographic evaluation in these cases. Palatal maxillary canine impaction in Class I uncrowded malocclusions was predicted on the basis of the absence of a permanent canine alveolar bulge superior to the deciduous canine, and on the frontal radiograph revealing a medially tipped permanent canine at the lateral border of the nasal cavity.23

Becker and colleagues25 reported that maxillary canine palatal impaction resulted from two events during canine eruption. The maxillary lateral incisor root was instrumental in both. Initially, the lateral incisor root failed to provide buccal guidance to the erupting canine, permitting its palatal movement. Once the maxillary canine was palatally positioned, the lateral incisor root impeded further canine eruption into the narrowing alveolus. Becker and colleagues25 demonstrated that people with absent or smaller-than-normal maxillary lateral incisors accounted for less than 1.1 percent of the population; however, 47.7 percent of people with palatally impacted maxillary canines had absent (5.5 percent) or small (42.2 percent) lateral incisors.

Small maxillary lateral incisors tend to possess shorter roots and develop later than normal.25 Therefore, they frequently fail to provide buccal guidance for erupting canines and then prevent further canine eruption into the narrowing alveolar process. Although there is no buccal guidance for an erupting maxillary canine when the lateral incisor is missing, canine eruption into the narrowing alveolar process is not prevented. Figure 2Go shows an 11-year-old girl with a haplodont (maxillary left lateral incisor), overretained deciduous maxillary left and right lateral incisors (both teeth firm to palpation), congenitally missing or unerupted maxillary right lateral incisor (no alveolar bulge noted) and the absence of maxillary canine buccal alveolar bulges. Neither the deciduous lateral incisor roots nor the undersized maxillary left lateral incisor provided sufficient buccal guidance for the erupting maxillary canines. In addition, the deciduous maxillary canines and maxillary lateral incisor prevented canine eruption into the narrowing alveolar process.



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Figure 2. Panoramic radiograph of an 11-year-old girl, which demonstrates a congenitally missing maxillary right lateral incisor, deciduous maxillary canines, lateral incisors with little or no root resorption, and mesially inclined and ectopically positioned maxillary canines with their crowns approximating the distal surface of the maxillary central incisor roots (suggestive of palatal impaction).

 
Genetic factors have been reported to be the primary cause of palatally impacted maxillary canines.26,27 An increased probability of palatally impacted canines is associated with other genetic dental anomalies (that is, tooth agenesis, smaller tooth size and delayed tooth development), bilateral occurrence (17 to 45 percent of cases), sex differences (as much as a 1:3.2 male-to-female ratio), familial history of palatally impacted canines and ethnic differences (5:1 European-to-Asian ratio).27 Therefore, anomalous or missing permanent maxillary lateral incisors and palatally impacted maxillary canines are considered covariables.27 Regardless of whether a missing or anomalous maxillary lateral incisor is a primary etiologic factor of, or a covariable with, palatally impacted maxillary canines, the clinician should be aware that these conditions have a greater probability of appearing together.

Ankylosis. Researchers have reported that from 1.3 to 14 percent of the population have experienced ankylosis of a primary molar.2831 Maxillary deciduous molars tend to ankylose at the time of, or before the eruption of, the first permanent molars, while the mandibular deciduous molars ankylose at about age 7 to 8 years.32 Most deciduous molars with infraocclusion show progression with time.29,33,34 Figure 3Go is a panoramic radiograph of a 9-year-old girl with ankylosed deciduous first molars and deciduous mandibular second molars.



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Figure 3. A. Panoramic radiograph of a 9-year-old girl reveals ankylosed deciduous first molars (apparently farther from the occlusal plane) and deciduous mandibular second molars. B. Panoramic radiograph of the same patient three years later. Note the ankylosis of the deciduous maxillary second molars and the greater infraocclusion of the deciduous first molars and deciduous mandibular second molars. The deciduous mandibular first molars exfoliated normally at a time consistent with the development of the dentition, the mandibular second molars are about to exfoliate and the deciduous maxillary molars are resorbing normally.

 
Reported consequences of deciduous molar ankylosis are loss of arch length, impaction of succedaneous teeth, ectopic eruption path of succedaneous teeth, supraeruption of opposing dentition, periodontal compromise to adjacent permanent teeth, surgical complication of deciduous tooth removal (trauma), introduction of epithelial tissue into the bony alveolus and attenuated alveolar growth.35,36 However, numerous longitudinal studies have revealed that infraoccluded molars that exfoliated naturally within a normal period did not affect the eruption time or path of succedaneous teeth, did not permanently affect alveolar bone development and did not adversely affect the root development of succedaneous teeth.29,31,34,37 In addition, Kurol and Thilander34 noted spontaneous uprighting of all permanent molars with the eruption of the premolars, which resolved the arch-length discrepancy resulting from the mesial tipping of the first molar. The removal of ankylosed deciduous molars is unwarranted unless the succedaneous tooth is erupting abnormally or the occlusal surface of the ankylosed deciduous molar is level with the gingival height of the adjacent teeth.32,35,37

Abnormal tooth bud position. Poorly rotated or angulated premolars often are associated with atypical root resorption of the deciduous molars.36 Figure 4Go is a panoramic radiograph of a 10-year-old patient, which reveals absence of third molars and the mandibular right second premolar, an atypically positioned mandibular left second premolar crown and insufficient maxillary and mandibular arch lengths. Radiography should be repeated about nine months later to determine if the position of the developing mandibular left second premolar has improved and if the mandibular right second premolar is beginning to develop. Interceptive treatment (that is, eruption guidance) also should be considered for arch-length discrepancies. According to Silling and colleagues,38 minor positional differences in a developing permanent premolar associated with a retained deciduous tooth correct themselves after the deciduous tooth is removed.



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Figure 4. Panoramic radiograph of a 10-year-old patient reveals the absence of third molars and the mandibular right second premolar, an atypically positioned mandibular left second premolar crown and insufficient maxillary and mandibular arch lengths.

 
Postimpaction observation. Attempting to reposition an impacted tooth into the alveolar process is not always warranted. Complications associated with treatment must be weighed against possible pathological concerns associated with observation. Dentists must consider each patient’s medical status, dental status, age, periodontal condition, hygiene, motivation, adverse habits and functional and occlusal relationships. Complications resulting from treatment include osseous defects, root fracture, root resorption, short root development, fractured roots, the need for endodontic therapy and periodontitis. Only treatment that minimizes injury to the dentition and periodontium should be considered. Treatment can be delayed until the patient is mentally and physically ready.

After tooth impaction is diagnosed and a decision not to treat is made, I recommend periodic observation to rule out possible pathological sequelae. During this postimpaction observation period, clinicians must perform clinical and radiologic examinations about every 18 to 24 months. The following pathological sequelae associated with tooth impaction have been noted: dentigerous cyst, odontogenic keratocyst, adenomatoid odontogenic tumor, calcifying epithelial odontogenic (Pindborg) tumor, odontogenic myxoma, ameloblastoma, ameloblastic fibroma, ameloblastic fibrous odontoma, ameloblastic fibrosarcoma (ameloblastic sarcoma), external/internal resorption of the impacted tooth, external root resorption of adjacent teeth, transmigration, referred pain and periodontitis.3949


   INTERVENTION
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
If a decision to treat is made, treatment should be as minimal as needed to facilitate natural eruption (the most physiologic type of tooth movement),50 permitting normal septal development and favoring the establishment of adequate bands of keratinized gingivae.7

Attempting to regain space lost from an ectopically erupting first molar is not always warranted.7 Efforts to regain space may be contraindicated in cases in which there is a congenitally missing tooth, a moderate-to-severe arch-length deficiency or a Class I molar relationship on the side of an ectopically erupting molar.

Space-regaining efforts require the maxillary first molar to move distally and bucally, rotate distally and become upright.51 Space can be regained through the placement of separators (brass, spring or elastic) between the maxillary deciduous second molar and the maxillary permanent first molar, with entrapment of 1 millimeter or less.6,7,51 With entrapment of 2 mm or more, the use of fixed or removable appliances is advised.7 Removal of the maxillary deciduous second molar is recommended only when it is not otherwise possible to regain arch length.7 If the deciduous molar has to be removed to regain space, then this space must be maintained after treatment.

Severe crowding, atypical resorption of the mandibular deciduous canine root by the mandibular permanent lateral incisor, or premature loss of one or both mandibular deciduous canines are symptoms of pronounced anterior arch-length discrepancies. After the mandibular incisors erupt, the mandibular arch-length discrepancy is predicted to determine the need for serial extraction. Serial extraction calls for the timed removal of primary teeth and, ultimately, permanent teeth.6 If necessary, serial extraction starts with the removal of primary incisors.6 Serial extraction simplifies and/or shortens orthodontic treatment by facilitating the eruption of the permanent teeth.6 However, when these patients are not properly observed or treated, serial extraction can cause tooth impaction.6

Although overretained or atypically resorbed deciduous teeth are considered to be a consequence, rather than a cause, of impaction,20 their removal is associated with improvement in, or correction of, permanent tooth position.20,39,52,53 The removal of a deciduous tooth is advised when there is radiographic evidence of atypical root resorption and the succedaneous tooth is unfavorably inclined or positioned. Fernandez and colleagues 54 recommended removal of the deciduous maxillary canine to prevent impaction when a panoramic radiograph revealed that the complete development of the maxillary lateral incisor and the overlapping of the canine with relation to the lateral incisor were associated with the absence of the canine bulge, lateral incisor agenesis, ankylosis, malformations or ectopic eruption.

The majority of impacted teeth erupt if hard- or soft-tissue obstructions are removed from their eruption paths.11,5558 Figure 5Go shows complete eruption of the mandibular incisors and delayed eruption of the maxillary incisors in a 10-year-old girl. During the course of a year, no change was noted in the position of the maxillary incisors, which were partially embedded in bone and covered by mucosa. The surgical procedure consisted of reflecting and apically repositioning a labial soft-tissue flap with keratinized tissue from the palate. Di Biase16 conducted a survey, which found that after the removal of supernumerary teeth, 75 percent of the impacted maxillary incisors spontaneously erupted. Many authors6,14,59 have reported that impacted teeth maintain the potential for eruption even after their root apexes form.



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Figure 5. A. Presurgical photograph of a 10-year-old girl demonstrates the complete eruption of the mandibular incisors and delayed eruption of the maxillary incisors. During the course of one year, no change occurred in the position of the maxillary incisors, which were partially embedded in bone and covered by mucosa. B. Photograph taken about three weeks after surgery to expose the maxillary incisors. The surgical procedure consisted of reflecting and apically repositioning a labial soft-tissue flap with keratinized tissue from the palate. Note the amount of incisor eruption during this period.

 
The majority of impacted teeth erupt if hard- or soft-tissue obstructions are removed from their eruption paths.


   RELOCATION
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
When interceptive treatment fails to improve the position or inclination of the developing tooth or when teeth are impacted, efforts to reposition them should be considered. Impacted teeth may be repositioned surgically or orthodontically.

Surgical relocation. Surgical repositioning (autotransplantation) is deemed a reasonable solution for treating impacted permanent teeth. Indications for surgical repositioning include the patient’s age, the need to minimize or eliminate orthodontic treatment and patient compliance.6063 The adult patient may not wish or be able to endure the rigors of extended orthodontic care. Minimizing or eliminating orthodontic treatment for patients with periodontally compromised teeth or marginal dental hygiene practices is recommended. Patients who are medically compromised or who have physical or mental disabilities may benefit from limited or no orthodontic treatment. Also, the cost of surgically assisted orthodontic treatment may be a limiting factor. Other factors to consider are presence or absence of adequate space, shape and status of the impacted tooth, and shape and status of adjacent teeth.60,62

Complications associated with autotransplanted teeth are devitalization, pulpal obliteration, decreased root length, periodontal compromises and root resorption.47,49,6372 These complications are affected by the extent of the transplanted tooth’s root development, the patient’s age, the tooth that is being transplanted, the ectopia of the impacted tooth, the length of extraoral storage during transplantation (that is, the time the tooth is out of the mouth) and the skill of the operator.6466,73

Orthodontic relocation. Kokich and Mathews39 recommend surgical exposure and orthodontic eruption of an impacted tooth when its apex is completely formed. Frequently, space must be orthodontically created before the clinician can surgically expose and orthodontically erupt an impacted tooth. This requires only one phase of orthodontic treatment and hastens the eruption of an impacted tooth.

The prognosis for orthodontically erupting and repositioning an impacted tooth within the alveolar process depends on the position and angulation of the impacted tooth, the length of treatment time, the patient’s age, the degree of patient cooperation, the available space and the presence of keratinized gingival tissue.6,74,75 The differential diagnosis for tooth impaction can be made only after the clinician surgically exposes the tooth and attempts orthodontic eruption. If tooth movement is not detectable with reasonable digital force, and orthodontic eruptive forces are unsuccessful, then the tooth is ankylosed. If tooth movement is perceived with reasonable digital force, but there is no response to orthodontic eruptive forces, then primary failure of eruption must be ruled out.

Complications associated with orthodontic repositioning of impacted teeth include absence of or inadequate keratinized gingivae, reduced sulcular depth, gingival recession, increased gingivitis, ankylosis, multiple exposures, devitalization, pulpal obliteration, external root resorption, injury to adjacent periodontium, marginal bone loss and extraction of the impacted tooth and/or an adjacent tooth.6,16,50,7581 Many of these complications can be reduced through judicious surgical exposure and appropriate orthodontic preparation of the dentition.

The orthodontic appliance (including cements, bands, brackets, auxiliaries, arch wires and ligature ties) and poor access to the crown of the impacted tooth compromise local hygiene practices.74,7678 Results of studies showing compromises to the periodontium of impacted and/or adjacent teeth are inconsistent. Generally, periodontal concerns in regard to impacted teeth have centered on the presence of adequate keratinized gingivae. Surgical techniques and eruption paths were designed to maintain or ensure the presence of adequate amounts of keratinized gingivae. Clinicians must weigh the possibility of creating periodontal problems involving an impacted tooth and/or adjacent teeth as a result of extensive osseous reduction against possible pulpal problems and mechanical challenges encountered while attempting to erupt an impacted tooth with limited osseous reduction.

Clinicians should consider only those treatment goals for impacted teeth that minimize injury to the dentition and periodontium.

Because of possible contamination by oral fluids, bonding an attachment or bracket to an impacted tooth is challenging. However, new bonding materials with an affinity for water can improve the placement of attachments on impacted teeth. These materials simplify bonding procedures, minimize the removal of bone from around the crown of the impacted tooth and have clinically acceptable bond strengths.82,83


   EXTRACTION
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
Clinicians should consider only those treatment goals for impacted teeth that minimize injury to the dentition and periodontium.50 Because it is not possible to reposition all impacted teeth within the alveolus, their removal may be indicated.50 The following factors need to be evaluated before an impacted tooth is extracted: patient’s age, dental status of adjacent teeth (including periodontal, endodontic and operative status, shape, resorption), dental status of the impacted tooth, occlusal relationship and arch length.39,55,84,85 Complications that are associated with the surgical removal of impacted teeth include periodontally compromised adjacent teeth, damage to adjacent teeth, root fracture, neuropathy, sinus involvement and osseous defect.59


   SUMMARY
 TOP
 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 
To make a differential diagnosis for an impacted tooth, clinicians must perform a clinical assessment of the patient’s condition. Informed consent should address treatment options for impacted teeth and associated treatment complications.

I have presented four treatment options for the management of impacted teeth: observation, intervention, relocation and extraction. Some interaction exists among the treatment options, and some options are viable only for a specific period. Before the dentist can recommend an appropriate treatment plan to a patient, he or she must perform a clear clinical and radiographic assessment of the patient’s mouth, review the pathological ramifications resulting from impacted teeth, discuss the complications of treatment with the patient and consider the patient’s expressed concerns.


   FOOTNOTES
 

Dr. Frank is a board-certified orthodontist in private practice. He also teaches part-time at the University of Florida, Jacksonville, and the Jacksonville Naval Air Station. Address reprint requests to Dr. Frank, 8657 Autumn Green Drive, Jacksonville, Fla. 32256.


   REFERENCES
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 ABSTRACT
 TREATMENT OPTIONS
 OBSERVATION
 INTERVENTION
 RELOCATION
 EXTRACTION
 SUMMARY
 REFERENCES
 

  1. Kramer RM, Williams AC. The incidence of impacted teeth: a survey at Harlem Hospital. Oral Surg Oral Med Oral Pathol 1970;29:237–41.[Medline]

  2. Grover PS, Lorton L. The incidence of unerupted permanent teeth and related clinical cases. Oral Surg Oral Med Oral Pathol 1985;59:420–5.[Medline]

  3. Dachi SF, Howell FV. A survey of 3874 routine full-mouth radiographs, II: a study of impacted teeth. Oral Surg Oral Med Oral Pathol 1961;14:1165–9.[Medline]

  4. Tilander B, Myrberg N. The prevalence of malocclusion in Swedish school children. Scand J Dent Res 1973;81:12–20.[Medline]

  5. Shah RM, Boyd MA, Vakil TF. Studies of permanent tooth anomalies in 7,886 Canadian individuals, I: impacted teeth. J Can Dent Assoc 1978;44:262–4.

  6. Proffit WR. Contemporary orthodontics. St. Louis: Mosby; 1993:217–9, 403–10.

  7. Kennedy DB, Turley PK. The clinical management of ectopically erupting first permanent molars. Am J Orthod Dentofacial Orthop 1987;92:336–45.[Medline]

  8. Harrison LM, Michal BC. Treatment of ectopically erupting permanent molars. Dent Clin North Am 1984;28:57–67.[Medline]

  9. Pulver F. The etiology and prevalence of ectopic eruption of the maxillary first permanent molar. ASDC J Dent Child 1968;35: 138–46.[Medline]

  10. Bjerklin K, Kurol J. Ectopic eruption of the maxillary first permanent molars: etiologic factors. Am J Orthod 1983;84:147–55.[Medline]

  11. Luten JR. The prevalence of supernumerary teeth in primary and mixed dentitions. J Dent Child 1967;34:346–53.[Medline]

  12. Howard RD. The unerupted incisor: a study of the postoperative eruptive history of incisors delayed in their eruption by supernumerary teeth. Dent Pract 1967;17:332–42.

  13. Primosch RE. Anterior supernumerary teeth: assessment and surgical intervention in children. Pediatr Dent 1981;3:204–15.[Medline]

  14. Nazif MM, Ruffalo RC, Zullo T. Impacted supernumerary teeth: a survey of 50 cases. JADA 1983;106:201–4.

  15. Bodenham RS. The treatment and prognosis of unerupted maxillary incisors associated with the presence of supernumerary teeth. Br Dent J 1967;123:173–7.[Medline]

  16. Di Biase DD. Mucous membrane and delayed eruption. Dent Pract Dent Rec 1971; 21:241–50.[Medline]

  17. Ericson S, Kurol J. Resorption of maxillary lateral incisors caused by ectopic eruption of the canines: a clinical and radiographic analysis of predisposing factors. Am J Orthod Dentofacial Orthop 1988;94:503–13.[Medline]

  18. Ericson S, Kurol J. Incisor resorption caused by maxillary cuspids: a radiographic study. Angle Orthod 1987;57:332–46.[Medline]

  19. Ericson S, Kurol J. Radiographic examination of ectopically erupting maxillary canines. Am J Orthod Dentofacial Orthop 1987;91:483–92.[Medline]

  20. Tilander B, Jakobsson SO. Local factors in impaction of maxillary canines. Acta Odontol Scand 1968;26:145–68.[Medline]

  21. Ericson S, Kurol J. Longitudinal study and analysis of clinical supervision of maxillary canine eruption. Community Dent Oral Epidemiol 1986;14:172–6.[Medline]

  22. Ericson S, Kurol J. Radiographic assessment of maxillary canine eruption in children with clinical signs of eruption disturbance. Eur J Orthod 1986;8(3):133–40.[Free Full Text]

  23. Williams BH. Diagnosis and prevention of maxillary cuspid impaction. Angle Orthod 1981;51:30–40.[Medline]

  24. Moss JP. The unerupted canine. Dent Pract Dent Res 1972;22:241–8.

  25. Becker A, Smith P, Behar R. Anomalous maxillary lateral incisors in relation to palatally displaced cuspids. Angle Orthod 1981;51:24–9.[Medline]

  26. Peck S, Peck L, Kataja M. The palatally displaced canine as a dental anomaly of genetic origin. Angle Orthod 1994;64:249–56.[Medline]

  27. Jacobs SG. The impacted maxillary canine: further observations on aetiology, radiographic localization, prevention/interception of impaction, and when to suspect impaction. Aust Dent J 1996;41:310–6.[Medline]

  28. Ben-Bassat Y, Brin I, Fuks AB. Occlusal disturbances resulting from neglected submerged primary molars. ASDC J Dent Child 1991;58:129–33.[Medline]

  29. Douglass J, Tinanoff N. The etiology, prevalence, and sequelae of infraclusion of primary molars. ASDC J Dent Child 1991;58: 481–3.[Medline]

  30. Via WF. Submerged deciduous molars: familial tendencies. JADA 1964;69:127–9.

  31. Kurol J, Koch G. The effect of extraction of infraoccluded deciduous molars: a longitudinal study. Am J Orthod 1985;87:46–55.[Medline]

  32. Messer LB, Cline JT. Ankylosed primary molars: results and treatment recommendations from an eight-year longitudinal study. Pediatr Dent 1980;2:37–47.[Medline]

  33. Biederman W, Chem B. The problem of the ankylosed tooth. Dent Clin North Am 1968:409–24.

  34. Kurol J, Thilander B. Infraocclusion of primary molars and the effect on occlusal development: a longitudinal study. Eur J Orthod 1984;6:277–93.[Abstract/Free Full Text]

  35. Andlaw RJ. Submerged deciduous molars: a review, with special reference to the rationale of treatment. Int Assoc Dent Child 1974;5:59–66.

  36. Burch J, Ngan P, Hackman A. Diagnosis and treatment planning for unerupted premolars. Pediatr Dent 1994;16:89–95.[Medline]

  37. Raghoebar GM, Boering G, Stegenga B, Vissink A. Secondary retention in the primary dentition. ASDC J Dent Child 1991;58: 17–22.[Medline]

  38. Silling G, Keller JF, Feingold M. Retained primary teeth: their effect on developing occlusions. ASDC J Dent Child 1979; 46:296–9.[Medline]

  39. Kokich VG, Mathews DP. Surgical and orthodontic management of impacted teeth. Dent Clin North Am 1993;37:181–204.[Medline]

  40. Bishara SE. Impacted maxillary canines: a review. Am J Orthod Dentofacial Orthop 1992;101:159–71.[Medline]

  41. Bishara SE. Clinical management of impacted maxillary canines. Semin Orthod 1998;4:87–98.[Medline]

  42. Kuftinec MM, Strom D, Shapira Y, Nahlieli O. A case report: bilateral transmigration of impacted mandibular canines. JADA 1995;126:1022–4.

  43. Bishara SE, Kommer DD, McNeil MH, Montagano LN, Oesterle LJ, Youngquist HW. Management of impacted canines. Am J Orthod 1976;69:371–87.[Medline]

  44. Wertz RA. Treatment of transmigrated mandibular canines. Am J Orthod Dentofacial Orthop 1994;106:419–27.[Medline]

  45. Bhaskar SN. Synopsis of oral pathology. St. Louis: Mosby; 1981:217–25.

  46. Waldron CA. Odontogenic cysts and tumors. In: Neville BW, Damm DD, Allen CM, Bouquot JE, eds. Oral and maxillofacial pathology. Philadelphia: Saunders; 1995: 493–540.

  47. Nordenram A. Impacted maxillary canines: a study of surgically treated patients over 20 years of age. Swed Dent J 1987; 11:153–8.[Medline]

  48. Stafne EC, Austin LT. Resorption of embedded teeth. JADA 1945;32:1003.

  49. Pogrel MB. Evaluation of over 400 autogenous tooth transplants. J Oral Maxillofac Surg 1987;45:205–11.[Medline]

  50. Vanarsdall RL, Corn H. Soft-tissue management of labially positioned unerupted teeth. Am J Orthod 1977;72:53–64.[Medline]

  51. Kurol J, Bjerklin K. Treatment of children with ectopic eruption of the maxillary first permanent molar by cervical traction. Am J Orthod 1984;86:483–92.[Medline]

  52. Leivesley WD. Minimizing the problem of impacted and ectopic canines. ASDC J Dent Child 1984;51:367–70.[Medline]

  53. Brickbauer GP. Preventive and interceptive orthodontics for the growing child. Int J Orthod 1988;26:19–22.[Medline]

  54. Fernandez E, Bravo LA, Canteras M. Eruption of the permanent upper canine: a radiologic study. Am J Orthod Dentofacial Orthop 1998;113:414–20.[Medline]

  55. Di Salvo NA. Evaluation of unerupted teeth: orthodontic viewpoint. JADA 1971; 82:829–35.

  56. Ohman I, Ohman A. The eruption tendency and changes of direction of impacted teeth following surgical exposure. Oral Surg Oral Med Oral Pathol 1980;49:383–9.[Medline]

  57. Clark D. The management of impacted canines: free physiologic eruption. JADA 1971;82:836–40.

  58. Shapira Y, Borell G, Kuftinec M, Nahlieli O. Bringing impacted mandibular second premolars into occlusion. JADA 1996;127:1075–8.

  59. Alling CC, Catone GA. Management of impacted teeth. J Oral Maxillofac Surg 1993;51:3–6.[Medline]

  60. Moss JP. An orthodontic approach to surgical problems. Am J Orthod 1975;68: 363–90.[Medline]

  61. Sagne S, Lennartsson B, Thilander B. Transalveolar transplantation of maxillary canines: an alternative to orthodontic treatment in adult patients. Am J Orthod Dentofacial Orthop 1986;90:149–57.[Medline]

  62. Lownie JF, Cleaton-Jones PE, Fatti P, Lownie MA. Autotransplantation of maxillary canine teeth: a follow-up of 35 cases up to 4 years. Int J Oral Maxillofac Surg 1986;15: 282–7.[Medline]

  63. Schatz JP, Joho JP. Indications of autotransplantation of teeth in orthodontic problem cases. Am J Orthod Dentofacial Orthop 1994;106:351–7.[Medline]

  64. Andreasen JO, Paulsen HU, Yu Z, Schwartz O. A long-term study of 370 autotransplanted premolars, III: periodontal healing subsequent to transplantation. Eur J Orthod 1990;12:25–37.[Abstract/Free Full Text]

  65. Schwartz O, Bergmann P, Klausen B. Autotransplantation of human teeth: a life-table analysis of prognostic factors. Int J Oral Surg 1985;14:245–58.[Medline]

  66. Andreasen JO, Paulsen HU, Yu Z, Bayer T. A long-term study of 370 autotransplanted premolars, IV: root development subsequent to transplantation. Eur J Orthod 1990;12:38–50.[Abstract/Free Full Text]

  67. Terry BC, Hegtvedt AK. Self-stabilizing approach to surgical uprighting of the mandibular second molar. Oral Surg Oral Med Oral Pathol 1993;75:674–80.[Medline]

  68. Paulsen HU, Andreasen JO, Schwartz O. Pulp and periodontal healing, root development and root resorption subsequent to transplantation and orthodontic rotation: a long-term study of autotransplanted premolars. Am J Orthod Dentofacial Orthop 1995;108: 630–40.[Medline]

  69. Schwartz O, Bergmann P, Klausen B. Resorption of autotransplanted human teeth: a retrospective study of 291 transplantations over a period of 25 years. Int Endod J 1985; 18:119–31.[Medline]

  70. Schatz JP, Joho JP. Autotransplantations and loss of anterior teeth by trauma. Endod Dent Traumatol 1993;9:36–9.[Medline]

  71. Cohen AS, Shen TC, Pogrel MA. Transplanting teeth successfully: autografts and allografts that work. JADA 1995;126:481–5.

  72. Schwartz O, Rank CP. Autotransplantation of cryopreserved tooth in connection with orthodontic treatment. Am J Orthod Dentofacial Orthop 1986;90:67–72.[Medline]

  73. Andreasen JO, Paulsen HU, Yu Z, Bayer T, Schwartz O. A long-term study of 370 autotransplanted premolars, II: tooth survival and pulp healing subsequent to transplantation. Eur J Orthod 1990;12:14–24.[Abstract/Free Full Text]

  74. Fournier A, Turcotte J, Bernard C. Orthodontic considerations in the treatment of maxillary impacted canines. Am J Orthod 1982;81:236–9.[Medline]

  75. Lorenz T. Orthodontic considerations of the impacted canine. J Gen Orthod 1990;1: 12–21.[Medline]

  76. Oliver RG, Hardy P. Practical and theoretical aspects of a method of orthodontic traction to unerupted teeth illustrated by three cases. Br J Orthod 1986;13:229–36.[Abstract]

  77. Woloshyn H, Kennedy DB, Joondeph DR. Pulpal and periodontal reactions to orthodontic alignment of palatally impacted canines. Angle Orthod 1994;64:257–64.[Medline]

  78. Darendeliler MA, Friedli JM. Treatment of an impacted canine with magnets. J Clin Orthod 1994;28:639–43.

  79. Boyd RL. Clinical assessment of injuries in orthodontic movement of impacted teeth, II: surgical recommendations. Am J Orthod 1984;86:407–18.[Medline]

  80. Becker A, Kohavi D, Zilberman Y. Periodontal status following the alignment of palatally impacted canine teeth. Am J Orthod 1983;84:332–6.[Medline]

  81. Gwinnett AJ, Ceen RF. Plaque distribution on bonded brackets: a scanning microscope study. Am J Orthod 1979;75:667–77.[Medline]

  82. Fricker JP. A new self-curing resin-modified glass-ionomer cement for the direct bonding of orthodontic brackets in vivo. Am J Orthod Dentofacial Orthop 1998;113:384–6.[Medline]

  83. Cacciafesta V, Jost-Brinkmann PG, Sussenberger U, Miethke RR. Effects of saliva and water contamination on the enamel shear bond strength of a light-cured glass ionomer cement. Am J Orthod Dentofacial Orthop 1998;113:402–7.[Medline]

  84. Wise RJ. Periodontal diagnosis and management of the impacted maxillary cuspid. Int J Periodontics Restorative Dent 1981;1:56–73.[Medline]

  85. Peterson LJ, Ellis E, Hupp JR, Tucker MR. Contemporary oral and maxillofacial surgery. St. Louis: Mosby; 1988:232–3, 246.





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